Summary of the etiology of renal decline

Summary of the etiology of renal decline
Renal side effects of the recession is very large, especially if patients without renal decline seize the time to heal and let condition deteriorated, then kidney recession may turn very bad for uremia high creatinine, thus causing irreparable harm to the patient, all for patients with renal decline, the understanding of the etiology of renal decline and make the relevant precautions are quite focused. So what is the etiology of renal decline concrete has it?
Here by my hospital for kidney specialists we explain in detail.
Chronic kidney recession reasons:
(1) The longer duration of diabetes, diabetic nephropathy patients, some people may exhibit renal complications, especially in young patients with type I diabetes, or may exhibit proteinuria PRO, which is often the first indication of renal involvement. Showed kidney disease patients with diabetes, about half in five years to produce impaired renal function, and other complications of diabetes, such as the reticuloendothelial system, vascular system and neurological complications may also exhibit simultaneously.
(2) is a congenital polycystic kidney disease, in the same family may have several patients disease. Check the pathogenesis of developmental defects can see a small water-filled balloon. Extrusion normal kidney cells. The disease can be long hidden until after 40 to 50 years old showed enlarged kidneys, significant renal dysfunction before the diagnosis of polycystic kidney disease.
(3) severe high blood pressure, severe high blood pressure can lead to renal arteriosclerosis, renal blood flow and less quantitative, impaired renal function. Renal artery disease itself, but also is the cause of loss of kidney function.
(4) lower urinary tract blockage, poor drainage of the lower urinary tract, such as benign prostatic hypertrophy, or certain anatomical defects caused by reflux of urine, causing renal pelvis, calyx expansion, called hydronephrosis, squeezing normal kidney cells, impaired renal function initiator.
(5) other very severe kidney disease caused less damage to normal, resulting in chronic renal failure. Because the body has a very strong compensatory ability, can appropriately adjust imbalances in vivo biochemical processes. Sometimes we can see only patients with normal renal function remaining 10%, and still sustain life.
(6) the renal glomerulus complex epidemic caused it to lose the pathogenesis of a variety of physiological changes resulting in glomerulonephritis, and ultimately lead to impaired renal function. Renal failure can show in a few months, a few years can be delayed until the serum urea and creatinine increased, decreased to half of normal kidney function, the performance will be exhibited. Sometimes after the onset of years, until after the terrible symptoms of uremia high creatinine only diagnosed with glomerulonephritis.
(7), interstitial nephritis, interstitial nephritis, the second largest incidence of chronic kidney decline. Tubular atrophy, fibrosis, scarring resulting in renal blood supply becomes less and renal dysfunction.
Analgesic-induced nephropathy, gouty nephropathy and kidney antibiotics and other toxic to the kidneys efficacy of drug-induced deputy belong interstitial nephritis.
Causes of acute renal decline mostly due to renal blood perfusion without feet, toxic nephritis or their primary kidney disease.
These are related to the pathogenesis of renal decline to explain the reasons for decline in renal patients, seize the time to go to regular hospital treatment is quite necessary. Read the above, it may be combined with their condition and treatment to make a judgment. If you have any questions, or around other kidney patients had a similar illness, and suggested that it might submit patient case information to our hospital with specific disease kidney specialist gives you detailed guidance and analysis. You can click on the icon to the right of the Experts Online slide, you can instantly and authority kidney specialist online communication, to get the most satisfactory answer!

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